Not long after birth, human babies transition from producing blood containing oxygen-rich fetal hemoglobin to blood bearing the adult hemoglobin protein. For children with sickle cell disease, the transition from the fetal to adult form of hemoglobin - the oxygen-carrying protein in blood - marks the onset of anemia and painful symptoms of the disorder. Now, new research led by Howard Hughes Medical Institute (HHMI) investigator Stuart H...
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